What are the symptoms of flaccid paralysis?

Acute flaccid myelitis (AFM) is a rare but serious condition that affects the nervous system, causing muscles to weaken….Symptoms

  • Sudden muscle weakness in the arms or legs.
  • Neck weakness.
  • Some other symptoms that patients may have include drooping eyelids or a facial droop, and difficulty swallowing or slurred speech.

Where does flaccid paralysis affect the body?

Flaccid paralysis may affect one or more limbs but more commonly affects the lower limbs. Because it is a lower motor neurone disease the reflexes are also absent and the muscles involved waste. These muscles may be very tender with muscle spasm in the early stages.

What are the causes of AFP?

The major clinical causes of AFP were GBS (56.8%), central nervous system (CNS) infection (4.3%), CNS tumors (5.1%), transverse myelitis (5%), myasthenia gravis (2 cases), hypokalemic paralysis (2 cases), and botulism (one case).

Which bacteria causes flaccid paralysis?

Botulism is a serious illness that causes flaccid paralysis of muscles. A neurotoxin, generically called botulinum toxin, causes botulism and the bacterium Clostridium botulinum (and rarely by C. butyricum and C. baratii) produces the neurotoxin.

Which disease is associated with flaccid paralysis?

Polio and other viruses The term acute flaccid paralysis (AFP) is often used to describe an instance with a sudden onset, as might be found with polio. AFP is the most common sign of acute polio, and used for surveillance during polio outbreaks.

What are the causes of flaccid paralysis?

Flaccid paralysis is a neurological condition characterized by weakness or paralysis and reduced muscle tone without other obvious cause (e.g., trauma). This abnormal condition may be caused by disease or by trauma affecting the nerves associated with the involved muscles.

What is the most common cause of acute flaccid paralysis?

The most common etiology of acute flaccid paralysis in this entire population was neuroparalytic snake envenomation, which was responsible for 51.9% of the cases, followed by the Guillain Barre syndrome (33.1%). These two etiologies accounted for 85% of all patients.

What diseases cause flaccid paralysis?

Conclusion: Neuroparalytic snakebite and Guillain Barre syndrome were the most common causes of acute flaccid paralysis in adults in our study.

Why does stroke cause flaccid paralysis?

Flaccid paralysis, a medical term for complete lack of voluntary movement, often sets in during Stage 1. This paralysis is caused by nerve damage that prevents the muscles from receiving appropriate signals from the brain, whether or not the brain is still capable of moving those muscles.

How is AFM spread in children?

Anyone of any age can get AFM, but it is more commonly reported in children. How is AFM spread? AFM is not spread from person to person. The viruses that are believed to cause AFM may be contagious from one person to another or may be spread by a mosquito or other vector depending on which virus causes the AFM.

Which is the best description of flaccid paralysis?

Flaccid Paralysis. Flaccid paralysis is a condition characterized by extreme weakness of muscles and loss of muscle tone. Acute paralysis in limbs that are flaccid (floppy or limp) are associated with this condition.

What kind of paralysis affects both arms and legs?

The type usually depends on where your brain or spinal cord is injured. Monoplegia is a kind of generalized paralysis that affects just one limb. Diplegia affects the same area on both sides, like both arms, both legs, or both sides of your face.

Can a lower motor neuron cause flaccid paralysis?

Flaccid paralysis can be associated with a lower motor neuron lesion. This is in contrast to an upper motor neuron lesion, which often presents with spasticity, although early on this may present with flaccid paralysis.

How is curare used to treat flaccid paralysis?

Curare. Curare acts as a neuromuscular blocking agent which induces flaccid paralysis. This poison binds to the acetylcholine (ACh) receptors on the muscle, blocking them from binding to ACh. As a result, ACh accumulates within the neuromuscular junction, but since ACh cannot bind to the receptors on the muscle, the muscle cannot be stimulated.